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Elimination of CXCR2 by gene targeting or blocking its activation having an antibody minimized neutrophil recruitment from the lung, lung edema and protein leakage [15, 37]. CXCR2 receptors on hematopoietic and non-hematopoietic cells are both essential for neutrophil recruitment in response to LPS, Every accounting for about 50 % of neutrophil rec